Thursday, November 12, 2015
Kiesselbach’s area.
The facial artery gives off the superior labial artery; the septal branch of which enters the nasal cavity through the nares and joins the anastomoses in Little’s area. The greater palatine artery, a terminal branch of the maxillary artery, passes through the greater palatine foramen and travels along the hard palate to enter the nasal cavity by way of the incisive canal, thus joining the anastomosis in Kiesselbach’s area.
Tuesday, November 10, 2015
facts
Patient Hygiene Performance (PHP) Index
The Patient Hygiene Performance (PHP) Index was first used as an oral hygiene assessment method when working with patients. Oral debris for the PHP Index is defined as soft foreign matter consisting of bacteria, mucin, and food loosely attached to tooth surfaces. Teeth are disclosed and a mouth mirror examination is made of specific teeth. The examiner mentally divides each tooth into five subsections and determines the presence or absence of oral debris on each of the subdivisions. Summing the values for each of the five subdivisions provides the oral debris score for the tooth. An overall patient hygiene performance score is tabulated by dividing the sum of the tooth scores by the number of surfaces charted.
Basement membrane (type IV) collagen
The collagen IV molecule is 400 nm long and consists of two alpha 1 (IV) and one alpha 2 (IV) chains. Their genes COL4A1 and COL4A2 form a transcription unit in which they are arranged head-to-head and connected by a short common promoter region.cotton wool appearance
pagets disease
Adenoid cystic carcinoma

Monday, November 2, 2015
Allodynia
Allodynia (pain due to a stimulus that does not usually provoke pain) and hyperalgesia (increased pain from a stimulus that usually provokes pain) are prominent symptoms in patients with neuropathic pain. Both are seen in various peripheral neuropathies and central pain disorders, and affect 15–50% of patients with neuropathic pain. Allodynia and hyperalgesia are classified according to the sensory modality (touch, pressure, pinprick, cold, and heat) that is used to elicit the sensation. Peripheral sensitisation and maladaptive central changes contribute to the generation and maintenance of these reactions, with separate mechanisms in different subtypes of allodynia and hyperalgesia. Pain intensity and relief are important measures in clinical pain studies, but might be insufficient to capture the complexity of the pain experience. Better understanding of afllodynia and hyperalgesia might provide clues to the underlying pathophysiology of neuropathic pain and, as such, they represent new or additional endpoints in pain trials.
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